Speech Therapy

About Post-Stroke Aphasia

Aphaisa is a common occurence after a stroke, where the reported prevalence ranges upto 1/3rd of the patients who are in the acute phase. During the first three months of the disorder, spontaneous recovery is at its peak. However, some improvement can even take place even later on. The rate of complete recovery can vary from 21-50%. Aphaisa’s initial severe stage usually predicts the language function. Within the first month after stroke, the doctor can made a valid prognosis of the disorder. Read on to know more about post-stroke aphasia.

About Post-Stroke Aphasia

An Overview of Post-Stroke Aphasia

This disorder is classified as non-fluent aphasias, including transcortical, isolation, Broca’s and global motor aphasias. Also, it is classfied as fluent aphasias, including anomic, conduction, transcortical sensory and Wernickea��s aphasias. Now, it is proven that different aphasia types have a distinctive recovery patterns. Global aphasia is conisdered to have the poorest prognosis among the different types of aphasia.

Also, Broca’s aphasics most frequently recovers towards the anomic phase. Wernickea��s aphasics tends to recover towards conduction or anomic aphasias. The type of aphasia which is a link between the evolutionary process of non-fluent and fluent aphaisas is anomic aphasia.

Aphasic Stroke Patients Susceptible to Depression

Even though clinical experience has revealed that communicative disorders may lead to the persistence and severity of depression, psychiatric evaluations in aphasic patients are very rare. In a study conducted by Astrom et al, it was reavealed that post-stroke aphaisa acts like an important indicator of depression in the acute stage. In this study, the association between depression and aphasia was present for three months, but no longer.

Another study found that nearly half of the hospitalized patients with aphaisa were under depression. No correlation between depression and aphasia could be established among long-term stroke survivors. Apart from these findings, the non-fluency of aphasia was linked to severity and frequency of PSD.

In post-stroke aphasic patients, the depression etiology is known to be multi-factorial. Coping and psychosocial factors linked with this disorder play a key role in the development of reactive depression. On the contrary, depression and aphasia can be just viewd as a mere coincident result of stroke happening from a brain lesion.

Other Functional and Clinical Correlates of Aphasia

Many studies have found no clear evidence of a link between age and prevalence of aphasia. One study found that old age indicated a poor prognosis in global aphasia. This disorder is now linked to the impairment in ADL and severity of motor deficits during the first few months post-stroke. The link is established due to the fact that aphasic patients may have larger lesions. These large lesions cause more language distortions as well as impairment in ADL.

The severity of aphasia, in the presence of of any motor deficits, does not in any ways influence the final result after cerebral infarction. The problem of analyzing intelligence levels in aphasic patients hinders the research design as well as the result interpretation. It is wrong to hold verbal intelligence tests in aphasic patients, although verbalization used to conduct the nonverbal tests are not yet determined.

Many studies on the correlation between severity and type of aphaisa and non-verbal cognitive disorder revealed contradictory results. The severity and presence of aphasia was linked to non-verbal test scores, although the type of aphasia was not. Another test, which used the similar method for measuring visuospatial ability and logical reasoning, found that the type of aphaisa was determinant in the poor performance in any non-verbal test. However, the severity of aphasia was not a determinant.

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